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These were the early indications in the postmodern era of cell-basedimmunotherapy for cancer that there was another force resisting this form of therapy. It turns out that both of these types of Tregs can perform this type ofDC inhibition, but the Tregs created from the anti-cancer T cell pool can do it even moreefficiently

It turns out that both of these types of Tregs can perform this type ofDC inhibition, but the Tregs created from the anti-cancer T cell pool can do it even moreefficiently.

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Kalo E et al (2007) Mutant p53 attenuates the SMAD-dependent transforming growth factorbeta1 (TGF-beta1) signaling pathway by repressing the expression of TGF-beta receptor typeII. Early evidence that transcrip-tion dysregulation may be one mechanism of mutant p53 included the nding thatActinomycin D, a potent inhibitor of transcription, blocked the reduction in apoptosisrate mediated by mutant p53 in response to treatment with doxorubicin or cisplatin [7].One of the rst studies to address the role of gene transcription in the mechanism ofaction of p53 GOF mutants was reported by Deb and colleagues in 2005 [21]

Early evidence that transcrip-tion dysregulation may be one mechanism of mutant p53 included the nding thatActinomycin D, a potent inhibitor of transcription, blocked the reduction in apoptosisrate mediated by mutant p53 in response to treatment with doxorubicin or cisplatin [7].One of the rst studies to address the role of gene transcription in the mechanism ofaction of p53 GOF mutants was reported by Deb and colleagues in 2005 [21]. Antagonism of histamine They effec-tively block histamine induced bronchocons-triction, contraction of intestinal and other smoothmuscle and triple response—especially wheal,flare and itch. (1996) Recognizing syncope: pitfalls and surprises.

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